قد يتعرض بعض المرضي لخطأ جراحي اثناء العمليات يتسبب في قطع عصب بطريقه غير مقصوده

في الفيديو شرحت كيف يقوم العصب باعاده تجديد نفسه و الحالات التي تسبب في شكوه المرضي  من الام في مكان الجرح

في هذه الحاله قامت المريضه بعمل ولاده قيصريه ونتيجه خطا جراحي بسيط غير مقصود , انقطع احد الاعصاب تحت الجلد والمسؤله عن 

الاحساس ما أدي الي زياده الحساسيه والاستقبال بشكل زاذد عن الطبيعي وشكوه المريضه من الام

ولدعم التشخيص المزعوم يقوم الطبيب بحقن مخدر موضعي مكان الجرح ما يؤدي لاستجابه فوريه والتخلص من الالم بشكل مؤقت

. **Nerve Cell Repair**:

   - When a nerve is injured, the body initiates a repair process to restore its function.

Here are some key points:

     - **Axon Degeneration**: Shortly after injury, the proximal and distal ends of the nerve separate.

Axonal sprouts form, allowing for potential nerve regeneration.

     - **Myelin Clearance**: Around the 7th day, macrophages (immune cells) clean up debris from axons and myelin.

     **Regeneration**: If the injury is at the axon's end, regeneration can occur. The distal segment disintegrates, and cytoplasmic elements accumulate, promoting nerve healing.

   Clinical Presentation**: Nerve damage may cause reduced function, numbness, tingling, pain, sensitivity changes, and muscle weakness.

    Diagnostic Procedures**: Electromyography, nerve conduction studies, pain assessment, and muscle strength evaluation.

   management**: Cryotherapy, exercise, neurorehabilitation, and surgery].

. **Wallerian Degeneration**:

   - Wallerian degeneration is an active process that occurs after nerve injury:

     - **Definition**: It's anterograde degeneration of the distal axon due to a nerve lesion.

     - **Timeline**: Typically occurs 7 to 21 days after the injury.

     - **Stages**:

  Axon Degeneration**: Separation of proximal and distal ends, followed by axonal sprout formation.

   Myelin Clearance**: Macrophages clear axonal and myelin debris.

       Regeneration**: If the axon's end is intact, granular disintegration occurs, allowing for nerve regeneration.

    Clinical Signs**: Reduced function, numbness, pain, and muscle weakness.

     Diagnostic Tools**: Electromyography, nerve conduction studies, and sensory tests.

   Management**: Cryotherapy, exercise, neurorehabilitation, and surgey